Tachycardia
Tachycardia, also called tachyarrhythmia, is a heart rate that exceeds the normal resting rate. In general, a resting heart rate over 100 beats per minute is accepted as tachycardia in adults. Heart rates above the resting rate may be normal or abnormal.
Complications
Tachycardia can lead to fainting.When the rate of blood flow becomes too rapid, or fast blood flow passes on damaged endothelium, it increases the friction within vessels resulting in turbulence and other disturbances. According to the Virchow's triad, this is one of the three conditions that can lead to thrombosis.
Causes
Some causes of tachycardia include:- Adrenergic storm
- Anaemia
- Anxiety
- Atrial fibrillation
- Atrial flutter
- Atrial tachycardia
- Atrioventricular reentrant tachycardia
- AV nodal reentrant tachycardia
- Brugada syndrome
- Circulatory shock and its various causes
- Dehydration
- Dysautonomia
- Exercise
- Fear
- Hypoglycemia
- Hypovolemia
- Hyperthyroidism
- Hyperventilation
- Inappropriate sinus tachycardia
- Junctional tachycardia
- Metabolic myopathy
- Multifocal atrial tachycardia
- Pacemaker mediated
- Pain
- Panic attack
- Pheochromocytoma
- Sinus tachycardia
- Sleep deprivation
- Supraventricular tachycardia
- Ventricular tachycardia
- Wolff–Parkinson–White syndrome
- Alcohol intoxication
- Stimulants
- Cannabis
- Drug withdrawal
- Tricyclic antidepressants
- Nefopam
- Opioids
Diagnosis
- 1–2 days: >159 beats per minute
- 3–6 days: >166 bpm
- 1–3 weeks: >182 bpm
- 1–2 months: >179 bpm
- 3–5 months: >186 bpm
- 6–11 months: >169 bpm
- 1–2 years: >151 bpm
- 3–4 years: >137 bpm
- 5–7 years: >133 bpm
- 8–11 years: >130 bpm
- 12–15 years: >119 bpm
- 15 years–adult: Tachycardia >100 bpm
Differential diagnosis
An electrocardiogram is used to classify the type of tachycardia. They may be classified into narrow and wide complex based on the QRS complex. Equal or less than 0.1s for narrow complex. Presented in order of most to least common, they are:- Narrow complex
- * Sinus tachycardia, which originates from the sino-atrial (SA) node, near the base of the superior vena cava
- * Atrial fibrillation
- * Atrial flutter
- * AV nodal reentrant tachycardia
- * Accessory pathway mediated tachycardia
- * Atrial tachycardia
- * Multifocal atrial tachycardia
- * Cardiac tamponade
- * Junctional tachycardia
- Wide complex
- * Ventricular tachycardia, any tachycardia that originates in the ventricles
- * Any narrow complex tachycardia combined with a problem with the conduction system of the heart, often termed "supraventricular tachycardia with aberrancy"
- * A narrow complex tachycardia with an accessory conduction pathway, often termed "supraventricular tachycardia with pre-excitation"
- * Pacemaker-tracked or pacemaker-mediated tachycardia
Sinus
The body has several feedback mechanisms to maintain adequate blood flow and blood pressure. If blood pressure decreases, the heart beats faster in an attempt to raise it. This is called reflex tachycardia. This can happen in response to a decrease in blood volume, or an unexpected change in blood flow. The most common cause of the latter is orthostatic hypotension. Fever, hyperventilation, diarrhea and severe infections can also cause tachycardia, primarily due to increase in metabolic demands.Upon exertion, sinus tachycardia can also be seen in some inborn errors of metabolism that result in metabolic myopathies, such as Glycogen [storage disease type V|McArdle's disease (GSD-V)]. Metabolic myopathies interfere with the muscle's ability to create energy. This energy shortage in muscle cells causes an inappropriate rapid heart rate in response to exercise. The heart tries to compensate for the energy shortage by increasing heart rate to maximize delivery of oxygen and other blood borne fuels to the muscle cells.
"In McArdle's, our heart rate tends to increase in what is called an 'inappropriate' response. That is, after the start of exercise it increases much more quickly than would be expected in someone unaffected by McArdle's." As skeletal muscle relies predominantly on glycogenolysis for the first few minutes as it transitions from rest to activity, as well as throughout high-intensity aerobic activity and all anaerobic activity, individuals with GSD-V experience during exercise: sinus tachycardia, tachypnea, muscle fatigue and pain, during the aforementioned activities and time frames. Those with GSD-V also experience "second wind", after approximately 6–10 minutes of light-moderate aerobic activity, such as walking without an incline, where the heart rate drops and symptoms of exercise intolerance improve.
An increase in sympathetic nervous system stimulation causes the heart rate to increase, both by the direct action of sympathetic nerve fibers on the heart and by causing the endocrine system to release hormones such as epinephrine (adrenaline), which have a similar effect. Increased sympathetic stimulation is usually due to physical or psychological stress. This is the basis for the so-called fight-or-flight response, but such stimulation can also be induced by stimulants such as ephedrine, amphetamines or cocaine. Certain endocrine disorders such as pheochromocytoma can also cause epinephrine release and can result in tachycardia independent of nervous system stimulation. Hyperthyroidism can also cause tachycardia. The upper limit of normal rate for sinus tachycardia is thought to be 220 bpm minus age.
Inappropriate sinus tachycardia
is a diagnosis of exclusion, a rare but benign type of cardiac arrhythmia that may be caused by a structural abnormality in the sinus node. It can occur in seemingly healthy individuals with no history of cardiovascular disease. Other causes may include autonomic nervous system deficits, autoimmune response, or drug interactions. Although symptoms might be distressing, treatment is not generally seen by clinicians as needed.Ventricular
Ventricular tachycardia is a potentially life-threatening cardiac arrhythmia that originates in the ventricles. It is usually a regular, wide complex tachycardia with a rate between 120 and 250 beats per minute. A medically significant subvariant of ventricular tachycardia is called torsades de pointes, which tends to result from a long QT interval.Both of these rhythms normally last for only a few seconds to minutes , but if VT persists it is extremely dangerous, often leading to ventricular fibrillation.
Supraventricular
This is a type of tachycardia that originates from above the ventricles, such as the atria. It is sometimes known as paroxysmal atrial tachycardia. Several types of supraventricular tachycardia are known to exist.Atrial fibrillation
is one of the most common cardiac arrhythmias. In general, it is an irregular, narrow complex rhythm. However, it may show wide QRS complexes on the ECG if a bundle branch block is present. At high rates, the QRS complex may also become wide due to the Ashman phenomenon. It may be difficult to determine the rhythm's regularity when the rate exceeds 150 beats per minute. Depending on the patient's health and other variables such as medications taken for rate control, atrial fibrillation may cause heart rates that span from 50 to 250 beats per minute. However, new-onset atrial fibrillation tends to present with rates between 100 and 150 beats per minute.AV nodal reentrant tachycardia
is the most common reentrant tachycardia. It is a regular narrow complex tachycardia that usually responds well to the Valsalva maneuver or the drug adenosine. However, unstable patients sometimes require synchronized cardioversion. Definitive care may include catheter ablation.AV reentrant tachycardia
requires an accessory pathway for its maintenance. AVRT may involve orthodromic conduction or antidromic conduction. Orthodromic conduction usually results in a narrow complex tachycardia, and antidromic conduction usually results in a wide complex tachycardia that often mimics ventricular tachycardia. Most antiarrhythmics are contraindicated in the emergency treatment of AVRT, because they may paradoxically increase conduction across the accessory pathway.Junctional tachycardia
Junctional tachycardia is an automatic tachycardia originating in the AV junction. It tends to be a regular, narrow complex tachycardia and may be a sign of digitalis toxicity.Management
The management of tachycardia depends on its type, whether or not the person is stable or unstable, and whether the instability is due to the tachycardia. Unstable means that either important organ functions are affected or cardiac arrest is about to occur. Stable means that there is a tachycardia, but it does not seem an immediate threat for the patient's health, but only a symptom of an unknown disease, or a reaction that is not very dangerous in that moment.Unstable
In those that are unstable with a narrow complex tachycardia, intravenous adenosine may be attempted. In all others, immediate cardioversion is recommended.Stable
If the problem is a simple acceleration of the heart rate that worries the patient, but the heart and the general patient's health remain stable enough, it is possible to correct it by a simple deceleration using physical maneuvers called vagal maneuvers. But, if the cause of the tachycardia is chronic, it would return after some time, unless that cause is corrected.Besides, the patient should avoid receiving external effects that cause or increase tachycardia.
The same measures as in unstable tachycardia can also be taken, with medications and the type of cardioversion that is appropriate for the patient's tachycardia.