Poxytrin


Poxytrins[Thromboxane A2|] or dihydroxy-E,''Z,E''-polyunsaturated fatty acids are PUFA metabolites that possess two hydroxyl residues and three in-series conjugated [double bond]s in an E,''Z,E'' cis–trans configuration. Poxytrins have platelet-inhibiting properties that are not found in isomers with three conjugated double bonds presenting in a different geometry. The unique E,''Z,E'' configuration in poxytrins may prove to be relevant in treating human conditions and diseases that involve pathological platelet activation.

Types

Poxytrins are metabolites of docosahexaenoic acid, arachidonic acid, and α-linolenic acid. Poxytrins derived from AA are termed linotrins.
Trivial nameFormulaPrecursor
protectin DX 10R,17S-dihydroxy-4Z,7Z,11E,13Z,15E,19Z-docosahexaenoic acidDHA
10R,17S-diHDHA10R,17S-dihydroxy-4Z,7Z,11E,13Z,15E,19Z-docosahexaenoic acidDHA
10S,17S-diHDHA10S,17S-dihydroxy-4Z,7Z,11E,13Z,15E,19Z-docosahexaenoic acidDHA
7-epi-MaR17S,14S-dihydroxy-4Z,8E,10Z,12E,16Z,19Z-docosahexaenoic acidDHA
8S,15S-diHETE8S,15S-dihydroxy-5Z,9E,11Z,13E-eicosatetraenoic acidAA
linotrin-19R,16S-dihydroxy-10E,12Z,14E-octadecatrienoic acidALA
linotrin-29S,16S-dihydroxy-10E,12Z,14E-octadecatrienoic acidALA

PDX

Protectin DX is perhaps the most prominent poxytrin. It is not to be confused with its isomer protectin D1. PD1 is structurally identical to PDX except that its three conjugated double bonds 11E,13E,15Z have the E,''E,Z'' configuration. PDX and PD1 both possess potent specialized pro-resolving mediator anti-inflammatory activity, but only PDX inhibits human platelet aggregation responses. PDX's anti-platelet activity is shared with various other dihydroxy-E,''Z,E''-PUFAs, but not with dihydroxy-PUFAs that have an E,''E,E'' or E,''E,Z'' configuration.
Cells make PDX by metabolizing DHA by double oxygenation with a 15-lipoxygenase to form the 10R,17S-hydroxperoxy intermediate which is reduced to its 10R,17S-hydroxyl product, PDX, probably by cytosolic glutathione peroxidase 1. Serial metabolism by two different lipoxygenases or by a lipoxygenase and a cytochrome P45 on a 1Z,4Z,7Z-PUFA may also make a 1,7-dihydroxy 2E,4Z,6E product.

Other poxytrins

Linotrins

Linotrin-1 and linotrin-2 are among the four isomeric metabolites produced by incubating ALA with ALOX15B. The extent to which the linotrins form in cells or in vitro is not clear.

Activity

Stimulating agents such as collagen depend on platelets to make and release
thromboxane A2
to mediate and/or enhance their aggregating activity. 10R,17S-diHDHA, and PDX to a slightly lesser degree, inhibit the human platelet aggregation response to collagen at ≥ 100–200 nanomolar concentrations. This appears to reflect the ability of poxytrins to inhibit the activities of COX-1 and COX-2, thereby blocking the production of TXA2 and thus interfering with the activation of the thromboxane receptor by TXA2. The linotrins appear to use a similar mechanism, and to have similar or slightly lower potencies. However, the linotrins are 20- to 100-fold stronger in inhibiting human platelet aggregation compared to 5-HETE and 12-HETE, two mono-hydroxyl-containing eicosanoids that contain an E,Z conjugated double bond configuration. Other biologically-active poxytrins have yet to be tested for, but are projected to possess anti-platelet activity.