Giredestrant


Giredestrant is an investigational oral selective [estrogen receptor degrader] developed by Genentech, a member of the Roche Group, for the treatment of estrogen receptor-positive, HER2-negative advanced breast cancer, as well as endometrial cancer, ovarian cancer, and other solid tumors.
It is a potent, nonsteroidal compound that antagonizes estrogen effects by competitively binding to both wild-type and mutant estrogen receptors with nanomolar potency. Giredestrant works by inducing an inactive conformation of the estrogen receptor ligand-binding domain and promoting proteasome-mediated degradation of the receptor protein.
As of May 2025, giredestrant is in clinical trials and has received Fast Track designation from the Food and Drug Administration for ER+, HER2-negative second- and third-line metastatic breast cancer.

Mechanism of action

Giredestrant is a nonsteroidal SERD that binds the estrogen receptor, including wild-type and mutant forms, inducing a conformational change in the receptor's ligand-binding domain. This promotes proteasome-mediated ER degradation, preventing ER-mediated signaling critical for tumor growth in ER+ breast cancer.
Compared to selective estrogen receptor modulators or aromatase inhibitors, giredestrant's degradation mechanism better addresses resistance in ESR1-mutated tumors.

Research

In clinical trials, giredestrant has been evaluated in patients with ER+, HER2-negative breast cancer and other solid tumors, including endometrial and ovarian cancers, including those with ESR1 mutations, both as a single agent and in combination with other therapies. Its orally bioavailable.

Breast Cancer

Other Indications

Safety and adverse effects

Giredestrant is generally well-tolerated, with a safety profile similar to endocrine therapies like fulvestrant. Common adverse events from the acelERA study and other trials include fatigue, nausea, arthralgia, diarrhea, hot flashes, headache, and musculoskeletal pain. ematologic effects, liver enzyme elevations, and ovarian cysts have been reported. Bradycardia was observed at higher doses but mitigated at the 30 mg dose used in pivotal trials.